Dr Rowe’s presentation discusses the use of scientific theory in driving clinical assessment and treatment through the use of individual patient neurophysiological, neuropsychological and psychological
Quantitative EEG is a direct measure of the electrical energies of the brain which are produced by synapses on the dendrites and cell bodies of cortical pyramidal cells. The network dynamics necessary for proper function of the brain can be disrupted following a traumatic brain injury and can persist for years following even apparently minor injuries with no associated loss of consciousness.
The QEEG data collected in STARTTS Neurofeedback clinic indicates that the excessive temporal lobe alpha and frontal lobe disturbances are two profiles seen most frequently in traumatized clients.
In the 1990s Prof. Yuri Kropotov started his research in the field of quantitative EEG and evoked potentials in normal subjects, in ADHD population and in neurological patients to whom intracranial electrodes were implanted for diagnosis and therapy.
An important task of the human central nervous system is to link sensory information to appropriate response. This is the defining characteristic of adaptive behaviour in humans. Such adaptability is presumed to be mediated by working memory systems that process and respond to detected stimuli according to experience, needs, context and intention, and underpin the capacity to realise goals and plans.
This presentation will review the research of Dr. Robert Coben with Autistic Children with a view to understanding his coherence training methodology. Dr. Gary Schummer has analysed and adapted this methodology in his own research with ADHD and Autistic Spectrum children. Drs. Coben and Schummer base their coherence training on the Neuro Rep analysis of Dr. William Hudspeth.
Since the first reports of neurofeedback treatment in ADHD in 1976 many studies have been carried out investigating the effects of neurofeedback on different symptoms of ADHD such as inattention, impulsivity and hyperactivity. This technique is also used by many practitioners, but the question as to the evidence-based level of this treatment is still unclear.
Phenotype evaluation of the EEG was proposed in a paper in 2005 published in Clinical Electroencephalography and Neurophysiology. The retrospective analysis of many decades of experience has since been actively evaluated, from published works focusing on basic neuroscience (J. Pop-Jordanov), and the work from Holland on medication response prediction (M. Arns).
Direct controlled, non-invasive and painless activation of the human brain has been achieved with the introduction of the transcranial magnetic stimulation (TMS) technique. Initial studies in the 1980's and 90's traditionally used single or paired-pulse stimuli to modulate the excitability of the motor cortex.
Abnormal profiles of polyunsaturated fatty acids in the plasma phospholipids were observed in patients with closed head injuries, indicating that the metabolic response to injury encompasses changes
This presentation is an introduction of a new protocol, its implications and its use with different disorders. Beta reset protocol was used successfully to treat refractory PTSD and refractory migraines,
An important task of the human central nervous system is to link sensory information to appropriate response. This is the defining characteristic of adaptive behaviour in humans. Such adaptability is
Traumatic Brain injury can manifest clinically in a wide variety of physical and mental presentations. The neuropathology and neuroanatomy underlying a number of these clinical manifestations are presented
After several months of neurofeedback, a young woman said, “I have never been more myself and never known less who I am.” Although her statement is exceptional, her experience is not. By its nature, neurofeedback affects the nature of those who train. In discussions of neurofeedback, we tend to focus on the alleviation of symptoms. This talk seeks to extend the discussion to the effects neurofeedback has not only on symptoms, but also over time, on personality and identity.
The quantitative electroencephalogram (qEEG) is becoming a viable assessment tool for neurofeedback practitioners. The qEEG information is obtained with comparisons to norm-referenced databases which can be overwhelming to many practitioners. To simplify understanding of the data and improve clinical applicability, we have developed a unique approach to the interpretation of the qEEG.
Research has shown inconsistent evidence for the use of Transcranial magnetic stimulation (TMS) being able to induce significant effects on behavioural measures of motor output in healthy subjects (Huang
An EEG review is performed, with simultaneous processing by any participant with dongle-less access to the WINEEG software, demonstrating the de-artifacting using ICA extraction and semi-automated epoch
This joint presentation will examine clinical cases using the theoretical framework and practical EEG analysis as taught by Jay Gunkelman. Steps in this analysis involve: Looking at raw EEG and determining its most prominent features; linking those features to presenting symptoms and pretreatment testing; and reviewing some derived analyses and data summaries, ie. Spectral Displays, Topographic Displays, and Coherence analyses, and their relationship to presenting symptoms.
Learning theory principles that contribute to details of application for the most effective neurofeedback training program will be presented. It has been established that many learning theory principles (classical conditioning, shaping, generalization, etc) are involved in the application known as neurofeedback. This talk is aimed to elaborate on the learning theory principles involved in the effective application of neurofeedback. Additionally, this talk provides the current evidence supporting the use of neurofeedback in the treatment of ADHD and recommendations on the implementation of neurofeedback in clinical practice.
AD/HD is one of the most common psychiatric disorders of childhood, affecting approximately 5% of primary school children. Almost all models of the disorder accept that the behavioural cluster which
Autism spectrum disorders (ASD) are a group of neurodevelopmental conditions that are characterized by social, communicative, and behavioural impairments. Although the neurobiological basis of ASD is
This presentation address will relate research findings from various disciplines to help understand and identify the many possible causal factors for ADHD, Depression and Anxiety, hopefully leading
• ANSA subcommittee established in 2009 to develop process of certification of neurofeedback for Australian professionals • Decision in 2010 to develop affiliation with the now‐named
In the last five years, there have been two very important breakthroughs in our understanding of EEG signals with implications for Neurodiagnostics and neurotherapy practice. The first of these is the work of Robert T. Knight, and his group at Stanford, that have shown the importance of high gamma signals through their work on intracranial electrocorticography. These workers have also shown phase amplitude coupling between high gamma, and alpha, in visual processing.The second is the work of Ali Mazaheri and Ole Jensen on the visual word processing area, and mismatch negativity. This work has shown clear diagnostic differences between ADHD and non ADHD subjects, in the control of attention and Alpha and Theta amplitude before and after mistakes.
Self-controlled energo-neuro-adaptive-regulation known as scenar therapy was developed in Russia to equip aeronauts with a method for treating illness and accidental injury with therapy that did not
In this talk, Richard Silberstein describes research findings examining the effects of dopamine depletion and dopamine agonists on brain functional connectivity. These findings are also discussed in
As practitioners, we may find ourselves ahead of the research in our implicit knowledge and belief in the efficacy of neurofeedback as an intervention option for improved brain-based functioning. There
Neuroplasticity ranks as one of the most exciting findings to emerge from the “Decade of the Brain” as it opened doors to a number of emerging interventions for altering brain functions.
They can either facilitate or inhibit learning by the brain during such operations. This is achieved via tonic regulation of a cortical region (“cortical sheet”) to either lower (increased
This presentation provides a brief description of the development of LENS and what is involved when treating clients with LENS. This treatment will be compared with more traditional forms of Neurofeedback.
This presentation reviews the historical development of symptom guided neurofeedback (NFB) and EEG / QEEG based NFB, and their more recent synthesis in clinical practice. In the late 1960s, Professor Barry Sterman founded the field of NFB with his discovery that NFB can control seizure disorders. From that point on, practitioners began using NFB to treat a wide variety of disorders, mostly tracking changes in symptoms to guide NFB. At the same time, university based research continued to correlate EEG patterns with symptoms. With the rise of Quantitative EEG analysis in the 1980s, it was proposed to use deviations from normative data to guide NFB. Both voltage based norms and coherence based norms were developed. Thus two schools of thought developed, and a certain rivalry emerged between the two.
During this session, Terry discusses • The principle of operation of tDCS; • The operation of the tDCS hardware; • Site selection; • Resources available;
Cardiac issues seen in monitoring channels (PVC, PAT, couplets... and Q-T interval elongation and sudden death). EOG and frontal lobe function (frontal eye fields) EMG chronic levels (headaches, TMJ, peripheral tension in over-arousal) CNS over-arousal and Electrodermal/thermal (myo-epithelial cells and sweat glands).
The brain is an information processing machine adjusting itself to the environment. Information processing can be defined as reducing uncertainty. It has been suggested that the brain developed from an evolutionary point of view once living creatures started moving around in a changing and thus uncertain environment.
Dr Natalie Challis has found that effective treatment of Neurobehavioural disorders require a 4 pillared approach: Brain – QEEG and an individualised Loreta Neurofeedback protocol, Body – The clinical application of functional neurology focuses on regulating the activity of the brain, which stimulates changes throughout the nervous system, Behaviour – Working with an educational specialist, and a psychologist, to reinforce the neurological change into behaviour and Balance – Both the gut and brain originate early in embryogenesis from the same clump of tissue, which divides during fetal development.
Considerable evidence exists to suggest that a variety if not all cortical systems can undergo some type of plastic reorganisation. Modulation of afferent input (sensory deprivation or sensory increase) to the cortical areas represents at least one factor that determines the type of reorganisation observed. This innate plastic response is probably determined to a certain extent by the central integrative state of the neurons and glial components of the functional projection networks involved.
Leptin is one of the key elements of communication between the brain and the body’s nutritional reserves. When there is excess calorie accumulation, in the form of increased fat, this adipokine principally sends the brain a signal to decrease food intake and increase energy expenditure. The metabolic and endocrine roles of leptin have been exhaustively studied; however, its effects on human cognition are less clear. Our group and others, in a body of research spanning two decades, have shown that both in rare patients with genetically-based leptin deficiency, as well as in the general population, leptin has multiple effects on the brain, impacting not only on mental functions related to food-processing, but also on overall cognition.
Previously Brown and Jamieson found that increased Mu rhythm (10-12Hz) localised at right BA6/BA13 differentiates elite from amateur table tennis players while viewing an elite opposing player. These cortical regions include nodes in both the dorsal attention network (identifying 'where' an object is in space relative to one’s body) and in the ventral attention network (identifying and selecting salient sensory information). These results suggest engagement of a timing mechanism by elite table tennis players regulating alpha oscillations relative to incoming stimuli, allowing for optimal efficiency in selecting salient information and, importantly, inhibiting irrelevant information.
HRV biofeedback produces an immediate increase in vagal modulation (cardiac risk marker) by guiding the user to adopt a slow rhythmic breathing pattern. A gap in the HRV biofeedback literature includes a lack of knowledge concerning the persistence of this increase in vagal modulation at baseline and under dynamic stress conditions. The current study aimed to assess the effect of 30 days of HRV biofeedback training on vagal modulation at baseline and during both orthostatic and mental stress.
Pain is frustratingly complex. Some people who have terrible injuries report very little pain while others develop terrible pain following a very minor event. This suggests that pain is not related only to the degree of physical injury and as such, other processes must contribute to the experience of pain. A growing body of evidence suggests that people in pain often have an altered perception of their body part. For example, it may feel too big or too small than its actual size. Further, people in pain have disruptions in the evaluation of incoming information from that painful body part and from the space surrounding it.
This presentation describes the case of a 17 year old female athlete who suffered two consecutive concussions, producing post-concussion headache symptoms. The athlete was assessed following the initial
Developmental Dyslexia is the most common learning disability diagnosed in children (Ziegler et al., 2008), estimated to affect between 5 to 17.5% (Shaywitz, 1998). Longitudinal studies reveal that children with dyslexia display persistently poorer literacy than peers even after intensive intervention (e.g. Shaywitz, 2003). Neuroimaging studies reveal underactivity in the left posterior language areas of the brain in both adults and children relative to controls during phonological tasks (Hoeft et al., 2006). Advances in the use of Neurotherapy for the treatment of a range of psychological conditions (e.g. in ADHD, see Arns et al., 2009), lend support for its use as a treatment tool for normalising language function.
Despite the prevalence and generally chronic course of Attention-Deficit/Hyperactivity Disorder (ADHD) throughout childhood, clinical diagnosis is dependent on traditional methods of behavioural observation with no routine biological testing to clarify the nature of the underlying neurological disorder or to inform relevant treatment. The current review briefly describes the unique neurological attributes of ADHD and the potential of neurophysiological data to inform diagnosis and assessment of ADHD and then presents a concerted effort to systematically identify all relevant primary research. After applying appropriate inclusion and exclusion criteria, 26 citations were considered eligible for this review.
Regardless of the kind of therapy we practice, the primary treatment goal with those who have suffered developmental trauma is affect regulation. Neurofeedback can teach brains how to quiet their own limbic eruptions and ease the pulses of fear, shame and rage that overtake these patients. Developmental trauma is, however, a relational disorder. At its core is the profound experience of motherlessness. This cannot be addressed by neurofeedback alone. With understanding, skill and neurofeedback, psychotherapists can begin to provide an experience of regulation that these patients have not had and with that an experience of having been mothered. This workshop will focus on what the therapist treating developmental trauma must know about the interplay of affect regulation and motherlessness, how neurofeedback can affect the therapy relationship for both patient and therapist and what to anticipate in the therapeutic relationship as you begin to quiet affect. We will touch on the latest developments in the neuroscience of trauma to better understand well- tested and newer protocols for those suffering the aftermath of developmental trauma.
This presentation will look at the lifeline of alpha theta training from birth to its current status. From the Menninger Clinic to the prevailing and varying approaches by Bill Scott, Tato Sokhadze,
EEG Neurofeedback Training (NFT) is a non-invasive neurophysiological technique, aimed at facilitating learned self-regulation of electrical activity of the brain. Beyond research into clinical applications of neurofeedback, a relative surge of interest into the methodology has led to attempts to apply EEG neurofeedback as a means to facilitate performance enhancement among non-clinical populations. One such domain is the enhancement of athletic performance and motor function. While significant attempts have been undertaken to investigate outcomes of sensori-motor rhythm (12-15Hz) modulation on aspects of motor performance, research exploring behavioural outcomes of EEG-NFT targeting the beta frequencies (15-20Hz) appear scarce.
There have been extraordinary gains in understanding brain dynamics in the last decade. Quantitative EEG and FMRI connectivity studies have done much to better characterise the neural networks that underpin consciousness, as well as different psychopathological states. These demonstrated abnormalities encourage contemplation of the possible therapeutic interventions that may assist in normalising brain function. The literature about the positive benefits of intervention often is based on case series highlighting substantial improvement. However, high quality trials and even the meta analyses of the most investigated disorder, ADHD, have tended to fail to provide the necessary empirical support, for neurofeedback as an intervention.
This lecture shows how trauma directly affects the developing brain, and specific brain functions responsible for attention, concentration, regulating emotions and engaging in satisfying relationships. Even after receiving excellent therapy many traumatised children and adults continue to feel chronically on edge, scared, agitated, collapsed and helpless. To deal with this they often try to cope with alcohol or drugs, while doctors prescribe a large variety of different medications that may make life more manageable but that also affect motivation and curiosity, and that rarely really resolve the problem.