Eating Disorders are often misunderstood and rarely examined from a scientific viewpoint. This presentation explains the neurochemical, genetic and environmental etiology of eating disorders and the treatment implications. Participants will learn assessment tools, have a more complete understanding of these disorders and have proven methods to help their patients. There are neurochemical changes that occur in patients with eating disorders. Anorexia Nervosa and Bulimia Nervosa have different chemicals that promote the respective disorders. Studies are now showing that binge eating can alter brain functioning and promote addiction-like properties in response to some foods.
Genetic predisposition may be a factor in the creation of eating disorders, but now scientists are showing that the gene expression can change. Histones are proteins that surround the gene. These proteins change gene expression. Our lifestyle, choices, and food consumption affects histones. It has been shown that consumption of high volumes of certain foods that are high fat/ sugar dense increase certain proteins that effects the brain reward system and enhances motivation for more resulting in binge-eating and cravings. This protein is a highly stable protein that can produce lasting changes in gene expression.
Depression and anxiety are commonly co-occurring with eating disorders. Studies are showing how disordered eating behaviours correlate with the creation of depression and anxiety in some patients. A widely used method for weight control has shown to actually create disordered eating, eating disorders and obesity. This method creates long term changes in circulating hormones that controls appetite and satiety. Treatment for eating disorders must change. The science is giving clinicians many more tools that can help cease this worldwide epidemic.